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The transfection of miR-191a mimics and inhibitors into TNF--induced IEC-6 cells confirmed that ZO-1 was the target gene of miR-191a

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Mice deficient in cellular glutathione peroxidase develop normally and show no increased sensitivity to hyperoxia

Results In total, 18 studies were identified across seven therapeutic categories regarding available treatments (Table 1)

SELENOP per se was reported to be in oxidized form for the uptake to take place (Shetty et al., 2018)

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