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Notes Supported by the NIH National Institute for Aging (R01 AG059011)

Copper can interact with p53, playing a crucial role in regulating cell cycle progression and apoptosis ( 2+ exposure induces oxidative stress response by increasing ROS and protein carbonyl compound (PC) levels, downregulating antioxidant superoxide anion (ASA), anti-hydroxyl radical (AHR), superoxide dismutase (SOD), catalase (CAT), and glutathione peroxidase (GSH-Px) activities, and reducing glutathione (GSH) content and mRNA expression levels ( Copper ion carriers produce reactive oxygen species upon binding to copper, inducing oxidative stress, damaging DNA, and causing cell cycle arrest ( 2+ and Cu 2+ , exerting effects in various cancer cell models

(E) In the tumor-specific CTLs, H3K18 lactylation induced by tumor cell produced lactic acid promotes circATXN7 expression, making tumor-specific CTLs sensitive to tumor-mediated AICD by binding to NF-B p65 subunit and masking the p65 nuclear localization signal motif, thereby sequestering it in the cytoplasm (Zhou et al., 2024)

TABLE 1 Summary of genes involved in salt stress in tomato

Thus, AA must be ingested by food (or supplements), particularly tomatoes, pineapples, watermelons and all citrus fruits (Banafsheh and Sirous, 2016)

Earlier-stage research and a narrower use case