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This is why you want to investigate this aggressively with a doctor who specialize in metabolic or mitochondrial disorders

CPT1A and metabolic diseases Patients with metabolic syndrome, obesity, and type 2 diabetes frequently have non-alcoholic fatty liver disease, hypertriglyceridemia, and other lipid metabolism abnormalities, which can be somewhat improved by increasing CPT1A expression ( CPT1A in vascular diseases Metabolism of endothelial cell depends on FAO to resist oxidative stress in the development of blood vessels, and CPT1A plays a crucial role in this process, offering a new potential target for the treatment of vascular-related diseases ( CPT1A in heart failure Heart failure is one of the top causes of death and disability around the world, but there is yet no safe and effective clinical therapy for heart failure ( Perhexiline was an effective anti-angina drug used in the last century, but was recalled by the manufacturer because of hepatotoxicity and peripheral neurotoxicity ( CPT1A in NAFLD The global obesity epidemic has dramatically increased the prevalence of non-alcoholic fatty liver disease (NAFLD), with no approved treatment now ( CPT1A in multiple sclerosis Inuits have a low prevalence of multiple sclerosis, possibly associated with in CPT1A P479L mutation ( CPT1A in renal fibrosis Renal fibrosis is a result of several types of chronic kidney diseases, and currently, the only treatment is to control blood pressure and blood sugar levels ( CPT1A and cancer Oxidative stress is the key element that causes prostate cancer to develop ( One characteristic of cancer is metabolic reprogramming, which provides tumor cells the basic elements for fast cell growth and maintains cell survival under hypoxic or nutrient-deficient conditions ( According to growing amounts of experimental evidence published in reputable journals in recent years, CPT1A may be a significant drug target for a number of cancer cells, including breast cancer ( CPT1A in breast cancer Breast cancer is the most prevalent and leading cause of cancer death among women globally, which has a poor prognosis, a high rate of recurrence and metastasis, and a high fatality rate ( Invasion and lymphangiogenesis in breast cancer cells can be inhibited by CPT1A knockdown, and CPT1A-null Human Dermal Lymphatic Endothelial Cells (HDLEC cells) consistently showed impaired invasion and lymphangiogenesis ( via acetyl-CoA-mediated Histone H3 lysine 9 acetylation (H3K9ac), which can be reversed by the addition of acetate ( Myc-overexpressing triple-negative breast cancer (TNBC) has a greater bioenergetic dependence on FAO, and CPT1A can be pharmaceutically inhibited to reduce energy metabolism in Myc-overexpressing TNBC cells and stop tumor growth in a xenograft model of Myc-overexpressing TNBC ( Besides, Chemotherapy (CT) and radiotherapy (RT) target rapidly dividing cells but still have significant normal tissue toxicity

This characteristic could be linked to the increased incidence of POI after chemotherapy 118

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