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To examine whether gpx genes regulate axonal fusion cell-autonomously, we generated touch neuron-specific GPX transgenes in gpx mutant background

For this purpose, SemD, and a point mutated version ( mut SemD), in which the 12 proline and arginine residues in the PRD1 and PRD2 motifs were replaced by valine and alanine residues, respectively, were tested for interaction with the SH3 domain of SNX9 (Fig

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