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Interestingly, both in vivo and in vitro studies have shown that the accumulation of -syn in the mitochondria of human DA neurons leads to decreased complex I activity, increased production of ROS, reduced substrate-specific respiration, and increased mitophagy [86, 87]

Specific copper transfer from the Cox17 metallochaperone to both Sco1 and Cox11 in the assembly of yeast cytochrome C oxidase

RohrmannSOvervadKBueno-de-MesquitaHBJakobsenMUEgebergRTjnnelandAet al

A aggregates and triggers a pathogenic cascade, with and secretases breaking down APP to produce A

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We further showed that FUS/TLS, U1-snRNP and CPSF-160 may indeed form a large complex (~1.8 MDa)