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Consequently, in NSCLC, the malfunction of MET initiates the subsequent PI3K-AKT-mTOR signaling pathway, culminating in increased PD-L1 expression
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Fragment C domain of tetanus toxin mitigates methamphetamine neurotoxicity and its motor consequences in mice
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A new role for c-Met activated PI3K axes is involved in the constitution of lamellipodial protrusions that are crucial for transmigration cells through endothelial cells
