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It was found that ALPPS surgery caused an increase in IHH secreted by activated HSCs, and activated the HH signaling pathway in hepatocytes to promote the accumulation of GLI1 in the nucleus, thereby promoting LR (Langiewicz et al., 2017

Wester, et al

Intriguingly, the inhibitory effects are mitigated by the treatment with UNC1999 (an inhibitor of H3K27me3) and 5-aza-2-deoxycytidine (a DNA methyltransferase inhibitor) (unpublished data) (58, 60), suggesting methionine or SAM reduces the production of inflammatory factor in association with the histone and DNA methylation modification of specific genes

C., Raggatt, L

J Cell Biol 183(5):795803 Matsuda N, Sato S, Shiba K, Okatsu K, Saisho K, Gautier CA et al (2010) PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

[73] have studied the relationship between IAP deficiency and IAP overproduction and their effect on tight-junction protein (TJP) levels and function