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As PINK1 stabilizes on the MOM and autophosphorylates, it influences Parkin by phosphorylating pre-existing ubiquitin molecules already conjugated to MOM proteins at Ser65 [48], by phosphorylating pSer65-Ub-bound Parkin [44], or by directly phosphorylating the Parkin in the absence of its initial encounter with pSer65-Ub [44, 49, 50]

doi: 10.3390/pharmaceutics13081291 61

Studies indicate that the prevalence of cognitive decline and mood disorders in COPD patients is typically two to three times higher than in healthy individuals[18]

Nat Cell Biol 22(4): 412-424

This protein acts as a myrosinase cofactor, steering reactions toward specific byproducts (Figure 5)

13 , e31 (2011)